The relationship between proinflammatory cytokine levels and fibrinolytic system in obese patients


GERENLİ M., Tuǧrul A., Demir M., ARIKAN E., GÜLDİKEN S., Azcan Ş.

Trakya Universitesi Tip Fakultesi Dergisi, cilt.25, sa.1, ss.44-51, 2008 (Scopus, TRDizin)

  • Yayın Türü: Makale / Tam Makale
  • Cilt numarası: 25 Sayı: 1
  • Basım Tarihi: 2008
  • Dergi Adı: Trakya Universitesi Tip Fakultesi Dergisi
  • Derginin Tarandığı İndeksler: Scopus, TR DİZİN (ULAKBİM)
  • Sayfa Sayıları: ss.44-51
  • Anahtar Kelimeler: Cytokines, Fibrinolysis, Insulin resistance, Obesity/complications
  • Trakya Üniversitesi Adresli: Evet

Özet

Objectives: The aim of this study was to investigate the relationship between proinflammatory cytokines (TNF-α and IL-6), and fibrinolytic system parameters (t-PA, and PAI-1) and insulin resistance in obese individuals. Patients and Methods: The study included 54 obese subjects (BMI ≥30 kg/m 2; 41 females, 13 males; mean age 33.5 years) and 30 non-obese healthy individuals (BMI <25 kg/m2; 19 females, 11 males; mean age 22.3 years). Fibrinogen levels were measured by the coagulometric method and the measurements of TNF-α, IL-6, t-PA and PAI-1 were carried out by the ELISA method. Results: Compared with non-obese subjects, obese individuals had significantly higher fibrinogen (p<0.01), PAI-1 (p<0.001), TNF-α (p<0.01), and IL-6 (p<0.001) levels, and significantly lower t-PA level (p<0.001) and t-PA/PAI-1 ratio (p<0.001). We also found an inverse relationship between TNF-α and t-PA levels (p=0.007) and t-PA/PAI-1 ratio (p=0.016) in obese individuals. The presence or absence of insulin resistance did not affect proinflammatory cytokines and fibrinolytic system parameters in obese individuals. Conclusion: Our findings indicate increased inflammatory cytokine levels especially in TNF-α level, and decreased fibrinolysis in obese individuals. These changes may contribute to atherosclerotic process independent from insulin resistance in obesity. ©Medical Journal of Trakya University. Published by Ekin Medical Publishing. All rights reserved.